Vit-D Lung Damage

A multi-step process:

Step 1

• Vitamin D is oil based
• accumlates over time
• Sticky oil

Step 2

• calcium cannot dissolve in water
• calcium sticks to Vitamin D

Step 3

• calcium acculates on cell walls
  • like a virus attaching
• calcium stimulates cells

Step 4

• stimulated cells hyper produce
  • DNA
  • RNA (Covid-19)
  • enzymes

Step 5

• enzymes break down membranes
• weaken the cell walls
• weaken the artery walls
  • the arteries begin to bleed

Step 6

• broken membranes bleed uncontrollable
• We hemorrhage to death
• Covid is a hemorrhagic disease
  • lungs bleed and we die.

Summary:

• Vitamin D > calcium > cell stimulation > enzyme activation > membrane degration > bleeding disorders
• Vitamin D excess can lead to bleeding disorders (hemorrhage)

Covid-19, a bleeding disorder

• Covid-19 death hppens when lungs hemorrhage
• Covid-19 binds to vitamin D receptors (ACE-2)
  • Vitamin D --> hemorrage (stroke)

----- Reference summary:

Article should mirror Vitamin D, E, K, A/rdid715

1. Glutamate stimulates cells
2. through the calcium mechanism
3. cell stimulation increases enzymes
   • tongue enzymes stimulate taste
     • MSG --> umami
     • Umami, " tastes like meat"
4. enzymes digest the cell wall
5. cell wall failure causes cell death
   • Cell damage, death with high glutamate levels
6. Excess glutamate allows high levels of calcium to enter the cell
7. This activates a number of enzymes
8. These enzymes go on to damage cell structures such as components of the cytoskeleton, membrane, and DNA
9. Lung membranes become damaged and bleed

Excitotoxicity

From Wikipedia, the free encyclopedia

----- archived November 23, 2020

In excitotoxicity, nerve cells suffer damage or death when the levels of otherwise necessary and safe neurotransmitters such as glutamate, α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), or N-methyl-D-aspartic acid (NMDA) become pathologically high resulting in excessive stimulation of receptors. For example, when glutamate receptors such as the NMDA receptor or AMPA receptor encounter excessive levels of the excitatory neurotransmitter glutamate significant neuronal damage might ensue. Excess glutamate allows high levels of calcium ions (Ca2+) to enter the cell. Ca2+ influx into cells activates a number of enzymes, including phospholipases, endonucleases, and proteases such as calpain. These enzymes go on to damage cell structures such as components of the cytoskeleton, membrane, and DNA.[1][2] In evolved, complex adaptive systems such as biologic life it must be understood that mechanisms are rarely, if ever, simplistically direct. For example, NMDA in subtoxic amounts induces neuronal survival to otherwise toxic levels of glutamate.[3][4]